Oral chelation of the biliary tract and circulatory system in CV disease - Quantum Medicine Update

Townsend Letter for Doctors and Patients,  Nov, 2002  by Paul Yanick, Jr.

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Nutraceuticals and vascular biology hold great promise in the prevention and treatment of cardiovascular (CV) disease. Clearly, food choices impact gene transcription, protein expression, and enzyme function to determine CV health. As explained in detail in the August-September 2002 TLfDP, discoveries of nutrient-gene interactions and gene expression shed new light on the complexity of heart disease.

In the April 2002 issue of the Journal of the American Nutraceutical Association (JANA), Mark C Houston, MD examined and defined the cell and molecular mechanisms leading to CV disease. This article clearly documents that we are entering an exciting era of cellular and molecular biology. The judicious use of naturally-occurring foods and specific herbal compounds with angiotensin-converting enzyme inhibitor activity, as calcium channel blockers, or with angiotensin receptor-blocking properties presents a foundation for the safe and rational treatment of CV disease. (1)

It is well known that oxidative stress, nutrient-gene interactions and gene expression can have positive or negative influences on human vascular biology. Hypertension is commonly initiated or perpetuated by endothelial dysfunction and vascular smooth muscle dysfunction of the autonomic nervous system (ANS). The increase in atherosclerosis and CV disorders is related to "empty calorie" overfeeding and the processing of our food supply. (2)

To remedy dietary deficiencies, our approach has been to use combinations of phytonutrients -- balanced to meridianorgan connections -- for optimal antioxidant protection and more blood pressure and CV reduction than isolated "bullet-type" nutritional supplements. Other studies have supported this approach as superior to the use of isolated nutrients. (3-11) Whole food concentrates, rich in a wide spectrum of nutrients, have also been documented to:

1. Increase serum and lymphocyte antioxidant levels, reducing oxidative stress (12-14)

2. Reduce weight with increased lean body mass (15,17)

3. Improve brachial artery flow-mediated vasodilation and improved endothelial function (22)

4. Improve arterial compliance, and reduce homocysteine and BP levels (23)

5. The intake of multiple minerals in a covalent, non-ionic state is more effective than magnesium alone in reducing blood pressure. (24-26)

Oral Chelation of the Biliary Tract

Atherosclerosis and CV disorders typically involve disturbances in hepatic cholesterol metabolism and detoxification through the liver's transulferation pathways. Excess cholesterol is excreted from the body mainly via the liver to bile either as free cholesterol or after conversion to bile acid. The regulation of hepatic cholesterol and bile acid metabolism and the basic mechanisms for the formation of cholesterol saturated bile should be addressed in any type of chelation protocol. The dual action of the liver in synthesizing new cholesterol or excreting old cholesterol from blood into bile is often forgotten in mainstream medicine practices.

Biliary sphincter of Oddi dysfunction, biliary dyskinesia, odditis, papillitis, post-cholecystectomy pain, right upper quadrant pain, biliary stasis, sludge or plaque in the biliary tract, and the migration and presence of flukes (Clonorchis sinensis) that promote intrahepatic ductal inflammation, proximal stasis, stone formation, and cholangitis. The result: an abnormal accumulation of free and esterified cholesterol and triglycerides and a disturbance of delicate feedback networks for the synthesis and homeostatic controls of cholesterol by the liver.

If bile flow is not appropriately coordinated or there is duodenitis, the increase in bile pressure can disturb the sphincter of Oddi (the muscular valve surrounding the exit of the bile duct and pancreatic duct) and cause bile and cholesterol to leak back into the bloodstream, resulting in Atherosclerosis and CV disorders. Papillary Stenosis, results in a backup of bile flow and stretching (dilation) of the bile duct has been documented by scans and various X-ray techniques. (18-21)

Observations on hundreds of cases with duodenitis and bilary tract dysfunction lead to development of a useful and effective system of therapy. My earlier foundational research reported a high incidence of duodenitis in the clinical population. (25-26) Duodenitis commonly results in disturbances of the gall bladder and intrahepatic biliary tract with a resultant backup of toxins (as transulferation pathways become compromised and deficient) (27-33) leading to xenobiotic excesses causing damage to human vascular biology and congesting the lymphatic system. As lymph nodes become swollen and congested with xenobiotics, they lose their ability to protect the body against infection. This is especially true when the deep lymph channels of the gut become congested. (34) Since lymph capillaries, unlike the blood, are very permeable to proteins and foreign toxins, dental toxins and infections, these factors slowly find their way into lymph channels and clog and choke off the lymph-generated immune responses in the gut and elsewhere in the body. Sixty to eighty percent of duodenitis cases have hidden pockets of infection where wisdom teeth have been extracted in the past, establishing a meridian block from jaw ostitis.